Specifications
Immunogen
Rat/Mouse Insulin + Proinsulin
Target
Alternative Names
INS1; insulin I; insulin-1; Ins-1; Ins2-rs1;
Product Background
Antigen Description
Insulin decreases blood glucose concentration. It increases cell permeability to monosaccharides, amino acids and fatty acids. It accelerates glycolysis, the pentose phosphate cycle, and glycogen synthesis in liver. Defects in insulin are the cause of familial hyperproinsulinemia.
Pathway
Adipogenesis, organism-specific biosystem; Aldosterone-regulated sodium reabsorption, organism-specific biosystem; Aldosterone-regulated sodium reabsorption, conserved biosystem; Developmental Biology, organism-specific biosystem; Diabetes pathways, organism-specific biosystem; Disease, organism-specific biosystem; IRS activation, organism-specific biosystem;
Citations
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Munkhjargal, M; Hatayama, K; et al. Glucose-driven chemo-mechanical autonomous drug-release system with multi-enzymatic amplification toward feedback control of blood glucose in diabetes. BIOSENSORS & BIOELECTRONICS 67:315-320(2015).
Zhou, FF; Sun, RL; et al. Myostatin Gene Mutated Mice Induced with Tale Nucleases. ANIMAL BIOTECHNOLOGY 26:169-179(2015).
COA
Certificate of Analysis
