Rabbit anti-Arabidopsis thaliana NPR1 (Middle region) Polyclonal Antibody

It is well established that salicylic acid (SA) plays a critical role in the transcriptional reprograming that occurs during the plant defense response against biotic and abiotic stress. In the course of the defense response, the transcription of different sets of defense genes is controlled in a spatio-temporal manner via SA mediated mechanisms. Interestingly, different lines of evidence indicate that SA interplays with reactive oxygen species (ROS) and glutathione (GSH) in stressed plants. In this review we focus on the evidence that links SA, ROS, and GSH signals to the transcriptional control of defense genes. We discuss how redox modifications of regulators and co-regulators involved in SA-mediated transcriptional responses control the temporal patterns of gene expression in response to stress. Finally, we examine how these redox sensors are coordinated with the dynamics of cellular redox changes occurring in the defense response to biotic and abiotic stress.

Nonexpressor of pathogenesis-related gene 1 (NPR1) codes for a transcription cofactor involved in the activation of systemic acquired resistance, a salicylic acid (SA)-dependent defense response. This work reports the cloning and characterization of two new genes, CnNPR1 and CnNPR3 from coconut, homologous to AtNPR1 of Arabidopsis thaliana. The cDNA deduced amino acid sequence contains the protein-protein interaction domains the BTB/POZ and ANKYRIN repeat domains, and a nuclear localization site (NLS). Phylogenetic analysis grouped CnNPR1 in a clade with AtNPR1 and CnNPR3 in a clade with AtNPR3, both reported genes of A. thaliana. Exogenous application of SA to coconut plantlets induced changes in the expression of CnNPR1 and CnNPR3 in leaf, stem and root tissues, providing evidence of their possible role in the signaling cascade leading to SAR in coconut palm. This is the first report on the cloning of putative key genes in the SAR-type defense mechanism in coconut palm.