Mouse Anti-Human NSE monoclonal antibody, clone 712

Plasma as a rapidly evolving technology has been succeeded to widely exploit in various industrial fields. We attempt to address the short- and long-time effects of seed priming with cold plasma in Cichorium intybus. The seeds were subjected to plasma (dielectric barrier discharge). The post-reactions of the plasma-primed seedlings were monitored in response to different concentrations (0, 2, and 10 mgl(-1)) of selenium nanoparticle (nSe). The plasma treatments enhanced seedling early growth in both shoot and roots. Besides, the simultaneous treatments of nSe of 2 mgl(-1) and plasma synergistically improved seedling growth (mean = 78%). The plasma treatments mitigated the nSe10-associated phytotoxicity. The plasma and/or nSe treatments induced the enzyme activities of catalase (mean = 35%) and peroxidase (mean = 30%). In a complementary experiment, the long-time effects of plasma priming were monitored in plants grown under soil condition. The seed priming with cold plasma led to significant increases in shoot fresh mass (mean = 32%) and root biomass (mean = 26.8%). Moreover, the plasma-primed seedlings produced higher numbers of flowers (mean = 41.5%) and enhanced flower fresh weights (mean = 24%). The findings underline this hypothesis that exposure to plasma may associate with the activation of plant defense machinery and long-time modification in plant growth and development.

Aim: In a previous study, low and high-normal arterial carbon dioxide tension (PaCO2) were not associated with serum neuron-specific enolase (NSE) in cardiac arrest survivors. We assessed the effect of PaCO2 on NSE in cerebrospinal fluid (CSF) and serum. Methods: This was a retrospective study. PaCO2 for the first 24 h was analysed in four means, qualitative exposure state (qES), time-weighted average (TWA), median, and minimum-maximum (Min-Max). These subgroups were divided into low (LCO2) and high PaCO2 (HCO2) groups defined as PaCO2 < 35.3 and PaCO2 > 43.5 mmHg, respectively. NSE was measured at 24, 48, and 72 h (sNSE(24,48,72) and cNSE(24,48,72)) from return of spontaneous circulation (ROSC). The primary outcome was the association between PaCO2 and the NSE measured at 24 h after ROSC. Results: Forty-two subjects (male, 33; 78.6%) were included in total cohort. PaCO2 in TWA subgroup was associated with cNSE(24,48,72), while PaCO2 in the other subgroup were only associated with cNSE(24). PaCO2 and cNSE in qES subgroup showed good correlation (r = -0.61; p < 0.01), and in TWA, median, and Min-Max subgroup showed moderate correlations (r = -0.57, r = -0.48, and r = -0.60; p < 0.01). Contrastively, sNSE was not associated and correlated with PaCO2 in all analysis. Poor neurological outcome in LCO2 was significantly higher than HCO2 in qES, TWA, and median subgroups (p < 0.01, p < 0.01, and p = 0.02). Conclusion: Association was found between NSE and PaCO2 using CSF, despite including normocapnic ranges; TWA of PaCO2 may be most strongly associated with CSF NSE levels. A prospective, multi-centre study is required to confirm our results.