Mouse Anti-Citrinin monoclonal antibody, clone CIT

Phased array over-the-horizon (OTH) radar usually needs to detect sea target and airborne target in different directions. Conventionally, the sea target and airborne target are detected sequentially with the coherent integration completely accomplished for each of them. However, the long coherent integration time (CIT) required by the sea target makes the time interval between two airborne target detections too long, which is not conducive to tracking airborne target. To overcome this problem, this paper proposes a new timeframe multiplexing scheme, in which several short-time detections of airborne target are inserted into a long CIT of sea target detection. The proposed scheme seeks to improve the detection frequency of airborne target while ensuring the detection performance of two types of target. However, due to the insertion of airborne target detection, the echo from sea surface is received discontinuously which means that the coherent integration of sea target is incomplete. To tackle it, a recovery algorithm based on the Hankel structured matrix is introduced, which recovers the slow-time domain signal of each range bin and thereby guarantees the detection performance of sea target not damaged. Numerical simulations prove the effectiveness of the proposed algorithm for signal recovery, and also verify that the proposed operating scheme has a shorter time interval for airborne target detection, while the detection performance of two targets are well guaranteed.

Background Neutrophil extracellular traps (NETs) are fibrous structures released from activated neutrophils. NET formation has been reported to be associated with acute respiratory distress syndrome (ARDS). However, there are no reports dealing with serial changes of NET formation in tracheal aspirate of ARDS patients. Case presentation We report three cases of ARDS. Case 1 is a 69-year-old man with necrotizing fasciitis of the buttocks, case 2 is a 49-year-old woman with extensive burns (80% of total body surface), and case 3 is a 73-year-old woman with severe bacterial pneumonia. We found abundant expression of citrullinated histone H3 (Cit-H3) and the formation of NETs at the onset of ARDS in all cases. The amounts of Cit-H3 and NETs decreased with the amelioration of respiratory failure in cases 1 and 2. In case 2, the amounts of Cit-H3 and NETs increased with aggravation of infection and respiratory status. In case 3, the abundant expression of Cit-H3 and NETs persisted; the patient did not recover from ARDS and eventually died. Cit-H3 and NETs were found in tracheal aspirates even if the patients had no direct injury to the lung as in cases 1 and 2. Conclusions In these three cases, the formation of NETs was observed in tracheal aspirate of patients with ARDS by either direct or indirect insults to the lung. The amount of NET formation changed dynamically over the clinical course of each patient.