Dark mesons at the LHC
JOURNAL OF HIGH ENERGY PHYSICS
Authors: Kribs, Graham D.; Martin, Adam; Ostdiek, Bryan; Tong, Tom
Abstract
A new, strongly-coupled dark sector could be accessible to LHC searches now. These dark sectors consist of composites formed from constituents that are charged under the electroweak group and interact with the Higgs, but are neutral under Standard Model color. In these scenarios, the most promising target is the dark meson sector, consisting of dark vector-mesons as well as dark pions. In this paper we study dark meson production and decay at the LHC in theories that preserve a global SU(2) dark flavor symmetry. Dark pions - like the pions of QCD - can be pair-produced through resonant dark vector meson production, pp -> rho(D) -> pi(D)pi(D), and decay in one of two distinct ways: gaugephobic, when pi D -> ff ' generally dominates; or gaugephilic, when pi(D) -> W + h, Z + h dominates once kinematically open. Unlike QCD, the decay pi 0 ->gamma gamma is virtually absent due to the dark flavor symmetry. We recast a vast set of existing LHC searches to determine the current constraints on (and future opportunities for) dark meson production and decay. When m rho D is slightly heavier than 2m pi D and rho +/-,0 kinetically mixes with the weak gauge bosons, the 8 TeV same-sign lepton search strategy sets the best bound, m pi D > 500 GeV. Yet, when only the rho 0 kinetically mixes with hypercharge, we find the strongest LHC bound is m pi D > 130 GeV, that is only slightly better than what LEP II achieved two decades ago. We find the relative insensitivity of LHC searches, especially at 13 TeV, can be blamed mainly on their penchant for high mass objects or large missing energy. Dedicated searches would undoubtedly yield substantially improved sensitivity. We provide a GitHub page to speed the implementation of these searches in future LHC analyses. Our findings for dark meson production and decay provide a strong motivation for model-independent searches of the form pp -> A -> B + C -> SM SM + SM SM where the theoretical prejudice is for SM to be a 3rd generation quark or lepton, W, Z, or h.
Serum adipokines play different roles in type I and II ketosis
ASIAN-AUSTRALASIAN JOURNAL OF ANIMAL SCIENCES
Authors: Shen, Liuhong; Zhu, Yingkun; Xiao, Jinbang; Qian, Bolin; You, Liuchao; Zhang, Yue; Yu, Shumin; Zong, Xiaolan; Cao, Suizhong
Abstract
Objective: This study was conducted to investigate the differences in several serum adipokines in perinatal dairy cows with type I and II ketosis, and the correlations between these adipokines and the two types of ketosis. Methods: Serum adiponectin (ADP), leptin (LEP), resistin, tumor necrosis factor-alpha (TNF-alpha), and interleukin-6 (IL-6) levels, and energy balance indicators related to ketosis were measured. Type I and II ketosis were distinguished by serum glucose (Glu) and Y values and the correlations between adipokines in the two types of ketosis were analyzed. Results: beta-Hydroxybutyric acid of type I ketosis cows was significantly negatively correlated with insulin (INS) and LEP and had a significant positive correlation with serum ADP. In type II ketosis cows, ADP and LEP were significantly negatively correlated, and INS and resistin were significantly positively correlated. Revised quantitative INS sensitivity check index (RQUICKI) values had a significantly positive correlation with ADP and had a very significant and significant negative correlation with resistin, TNF-alpha, and IL-6. ADP was significantly negatively correlated with resistin and TNF-alpha, LEP had a significantly positive correlation with TNF-alpha, and a significantly positive correlation was shown among resistin, IL-6, and TNF-alpha. There was also a significant positive correlation between IL-6 and TNF-alpha. Conclusion: INS, ADP, and LEP might exert biological influences to help the body recover from negative energy balance, whereas resistin, TNF-alpha, and IL-6 in type II ketosis cows exacerbated INS resistance and inhibited the production and secretion of ADP, weakened INS sensitivity, and liver protection function, and aggravated ketosis.