Human HDV IgM ELISA Kit (DEIA072)

Regulatory status: For research use only, not for use in diagnostic procedures.

Write a review

Size
96T
Sample
serum, plasma
Species Reactivity
Human
Intended Use
The Human HDV IgM ELISA Kit is an enzyme-linked immunosorbent assay for qualitative determination of IgM-class antibodies to hepatitis D virus in human serum or plasma.
Contents of Kit
1. Microwell Plate
2. Negative Control
3. Positive Control
4. HRP-Conjugate Reagent
5. Sample Diluent
6. Stock Wash Buffer
7. Chromogen Solution A
8. Chromogen Solution B
9. Stop Solution
Storage
The components of the kit will remain stable through the expiration date indicated on the label and package when stored at 2-8°C, do not freeze. For more detailed information, please download the following document on our website.
Sensitivity
Sensitivity: 100%.

Citations


Have you cited DEIA072 in a publication? Let us know and earn a reward for your research.

Customer Reviews


Write a review, share your experiences with others and get rewarded !
Product Name Cat. No. Applications Host Species Datasheet Price Add to Basket
Product Name Cat. No. Applications Host Species Datasheet Price Add to Basket

References


Hydrotreating Reactivities of Atmospheric Residues and Correlation with Their Composition and Properties

ENERGY & FUELS

Authors: Cui, Qingyan; Ma, Xiaoliang; Nakano, Koji; Nakabayashi, Koji; Miyawaki, Jin; Al-Mutairi, Adel; Marafi, Abdulazim M. J.; Park, Joo-Il; Yoon, Seong-Ho; Mochida, Isao

In order to better understand the effects of composition and properties of atmospheric residues (AR) on their reactivities for hydrodesulfurization (HDS), hydrodenitrogenation (HDN), hydrodevanadium (HDV), hydrodenickel (HDNi), hydrodeasphaltene (HDAsp), and hydrodeconradson-carbon-residue (HDCCR) in the hydrotreating process, analysis and characterization of three ARs (AA-AR, AB-AR, and AM-AR) from Arabia crudes were conducted, and their hydrotreating reactivities were evaluated in a pilot unit over a catalyst system at 370 degrees C under a H-2 pressure of 13.5 MPa by comparing the conversions of the various species and their rate constants on each catalyst. The overall reactivity of various species decreases in the order of vanadium species > sulfur species approximate to asphaltenes > nickel species > Conradson carbon residue precursor > nitrogen species, regardless of the sources of the ARs. Reactivities of the three ARs in HDS, HDV, and HDAsp increase in the order of AB-AR < AA-AR < AM-AR, while reactivities of the three ARs in HDNi, HDCCR, and HDN are similar. The higher nitrogen and asphaltenes concentrations and larger density of AR have strong and negative effects on the HDS, HDV, and HDAsp reactivities but no significant effect on the HDN, HDNi, and HDCCR reactivities. The B parameter obtained from electron spin resonance analysis can be a good index to predict the HDV reactivity of A.R.

Innate immune recognition and modulation in hepatitis D virus infection

WORLD JOURNAL OF GASTROENTEROLOGY

Authors: Jung, Stephanie; Altstetter, Sebastian Maximilian; Protzer, Ulrike

Hepatitis D virus (HDV) is a global health threat with more than 15 million humans affected. Current treatment options are largely unsatisfactory leaving chronically infected humans at high risk to develop liver cirrhosis and hepatocellular carcinoma. HDV is the only human satellite virus known. It encodes only two proteins, and requires Hepatitis B virus (HBV) envelope protein expression for productive virion release and spread of the infection. How HDV could evolve and why HBV was selected as a helper virus remains unknown. Since the discovery of Na+-taurocholate co-transporting polypeptide as the essential uptake receptor for HBV and HDV, we are beginning to understand the interactions of HDV and the immune system. While HBV is mostly regarded a stealth virus, that escapes innate immune recognition, HBV-HDV coinfection is characterized by a strong innate immune response. Cytoplasmic RNA sensor melanoma differentiation antigen 5 has been reported to recognize HDV RNA replication and activate innate immunity. Innate immunity, however, seems not to impair HDV replication while it inhibits HBV. In this review, we describe what is known up-to-date about the interplay between HBV as a helper and HDV's immune evasion strategy and identify where additional research is required.

Online Inquiry

Name:
Phone: *
E-mail Address: *
Technology Interest:
Type of Organization:
Service & Products Interested: *
Project Description:
Verification code
Click image to refresh the verification code.

Online Inquiry

  Interested in larger quantities ? request a quote!
  Protocol may be improved. Please feel free to contact us to obtain the latest version.!

Ordering Information

Payment methods we support:
Invoice / Purchase Order
Credit card

OUR PROMISE TO YOU Guaranteed product quality expert customer support

Inquiry Basket