Human CCL17 (TARC) ELISA Kit (DEIA7849)

Regulatory status: For research use only, not for use in diagnostic procedures.

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Size
96T
Sample
serum, plasma, cell culture supernate, tissues lysates, body fluids
Species Reactivity
Human
Intended Use
The Human CCL17/TARC ELISA Kit is intended for detection of Human CCL17/TARC.
Contents of Kit
1. Lyophilized recombinant human CCL17 standard: 10 ng/tube x 2
2. One 96-well plate precoated with anti-human CCL17 antibody
3. Sample diluent buffer: 30 mL
4. Biotinylated anti-human CCL17 antibody: 130 μL
5. Antibody diluent buffer: 12 mL
6. Avidin-Biotin-Peroxidase Complex (ABC): 130 μL
7. ABC diluent buffer: 12 mL
8. TMB color developing agent: 10 mL
9. TMB stop solution: 10 mL
Storage
If used frequently, reagents may be stored at 2-8°C. If used infrequently, reagents should be stored at -20°C. For more detailed information, please download the following document on our website.
Detection Range
31.2 pg/mL-2000 pg/mL

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References


Facial Edema in an Elderly Man: An Unusual Presentation of Nonepisodic Angioedema with Eosinophilia

CASE REPORTS IN DERMATOLOGY

Authors: Hashimoto, Takashi; Muneta, Kanako; Watanabe, Ken

Nonepisodic angioedema with eosinophilia (NEAE) is a rare allergic disease with a young Japanese and East Asian female predominance. NEAE features transient, nonrecurrent angioedema and peripheral blood eosinophilia without visceral organ involvement. Angioedema in NEAE occurs on the extremities, while the trunk and face are rarely involved. Here, we report a case of NEAE affecting only the face in an 80-year-old Japanese man. He was otherwise healthy and took no medication until the sudden development of angioedema on the face. The extremities and trunk were not involved. Skin biopsy examination revealed eosinophilic infiltration and degranulation between collagen bundles through the entire dermis, and perivascular and perifollicular infiltration of eosinophils and lymphocytes, but no evidence of vasculitis. Peripheral hypereosinophilia and high serum thymus-and activation-regulated chemokine (TARC) level were noted. Visceral organ involvement, parasite infection, and an allergic response were not detected in the patient. Administration of oral corticosteroid improved his symptoms rapidly and dramatically with improvements in the blood eosinophil count and serum TARC level. After the corticosteroid was discontinued, no recurrence was observed for 3 years. Thus, he was diagnosed as having NEAE. It should be noted that angioedema with eosinophilia might occur with an unusual presentation and might develop in elderly patients. (c) 2017 The Author(s) Published by S. Karger AG, Basel

The Roles of Genetic Factors in Kawasaki Disease: A Systematic Review and Meta-analysis of Genetic Association Studies

PEDIATRIC CARDIOLOGY

Authors: Xie, Xiaochuan; Shi, Xiaohan; Liu, Meilin

This systematic review and meta-analysis aimed to better elucidate the roles of genetic factors in Kawasaki disease (KD), and determine the potential genetic biomarkers of KD. The systematic literature search of PubMed, Medline, Embase, Web of Science and CNKI identified 164 eligible studies. The qualitative synthesis revealed that 62 genes may be correlated with the susceptibility to KD, and 47 genes may be associated with the incidence of coronary artery lesions (CALs) in KD. A total of 53 polymorphisms in 34 genes were investigated in further quantitative synthesis. Of these, 23 gene polymorphisms were found to be significantly correlated with KD susceptibility, and 10 gene polymorphisms were found to be significantly associated with the incidence of CALs in KD. In conclusion, our findings indicate that gene polymorphisms of ACE, BLK, CASP3, CD40, FCGR2A, FG beta, HLA-E, IL1A, IL6, ITPKC, LTA, MPO, PD1, SMAD3, CCL17 and TNF may affect KD susceptibility. Besides, genetic variations in BTNL2, CASP3, FCGR2A, FGF23, FG beta, GRIN3A, HLA-E, IL10, ITPKC and TGFBR2 may serve as biomarkers of CALs in KD.

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