HSV 1+2 IgG, IgM ELISA Kit (DEIA05713)

Regulatory status: For research use only, not for use in diagnostic procedures.

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Species Reactivity
Intended Use
The HSV 1+2 tests are enzyme immunoassays (EIA) for the semi-quantitative determination of IgG or IgM antibodies against Types 1 and 2 of the Herpes simplex virus (HSV 1 and HSV 2) in human serum.
Contents of Kit
1. Microwell Plate
2. Sample Buffer
3. Wash Buffer
4. Standard Control IgG
5. Standard Control IgM
6. Negative Control IgG
7. Negative Control IgM
8. Control A IgG
9. Control B IgG
10. Control A I gM
11. Control B IgM
12. Anti-human IgG conjugate(goat)
13. Anti-human IgM conjugate(goat)
14. Substrate
15. Stop reagent
The test kit must be stored at 2-8°C and can be used until the expiry date printed on the label. For more detailed information, please download the following document on our website.
IgG-97.8 %
IgM-100.0 %


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RETRACTION: RGB and HSV quantitative analysis of autofluorescence bronchoscopy used for characterization and identification of bronchopulmonary cancer (Retraction of Vol 5, Pg 3023, 2016)


Authors: Zheng, X.; Xiong, H.; Li, Y.; Han, B.; Sun, J.

A 3D human brain-like tissue model of herpes-induced Alzheimer's disease


Authors: Cairns, Dana M.; Rouleau, Nicolas; Parker, Rachael N.; Walsh, Katherine G.; Gehrke, Lee; Kaplan, David L.

Alzheimer's disease (AD) is a neurodegenerative disorder that causes cognitive decline, memory loss, and inability to perform everyday functions. Hallmark features of AD-including generation of amyloid plaques, neurofibrillary tangles, gliosis, and inflammation in the brain-are well defined; however, the cause of the disease remains elusive. Growing evidence implicates pathogens in AD development, with herpes simplex virus type I (HSV-1) gaining increasing attention as a potential causative agent. Here, we describe a multidisciplinary approach to produce physiologically relevant human tissues to study AD using human-induced neural stem cells (hiNSCs) and HSV-1 infection in a 3D bioengineered brain model. We report a herpes-induced tissue model of AD that mimics human disease with multicellular amyloid plaque-like formations, gliosis, neuroinflammation, and decreased functionality, completely in the absence of any exogenous mediators of AD. This model will allow for future studies to identify potential downstream drug targets for treating this devastating disease.

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